Author

Mizuki Azuma

University of KansasORCID 0000-0001-9411-9130Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Assay:proteomics· 1

Frequent Co-Authors

Alejandro Parrales· 1 shared Evan T. Schulz· 1 shared Haeyoung Kim· 1 shared Harsha HapugaswattaiD· 1 shared Hyewon Park· 1 shared Tomoo IwakumaiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

6 top collaborators · 10 mutual co-author links

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1 shared paperCancer Biology

Alejandro Parrales

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJun 19, 2026

A combination of EWSR1-FLI1 with loss of one EWSR1 allele promotes damage in the mitotic spindle and sensitization to microtubule-destabilizing agent

Evan T. Schulz, Mizuki Azuma

The paper shows that combining EWSR1-FLI1 expression with EWSR1 knockdown increases mitotic spindle damage and sensitivity to microtubule-destabilizing agents in Ewing sarcoma cells.

DOI 10.64898/2026.06.15.732411

humanproteomics

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Cancer BiologybioRxivPreprintMay 24, 2026

The combination of EWSR1-FLI1 and loss of one EWSR1 allele leads to the induction of trisomy 8

Harsha Hapugaswatta, Alejandro Parrales, Hyewon Park…·Mizuki Azuma

This paper shows that EWSR1-FLI1 fusion and loss of one EWSR1 allele induce trisomy 8 through disruption of the EWSR1-Aurora B pathway.

1 citation

humanexperimental assay

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