Jin‐Mo Kim

This study shows that muscarinic signaling in the prefrontal cortex compensates for declining thalamofrontal communication during short-term memory by enhancing neuronal excitability.

This study shows that adolescent NMDAR hypofunction impairs short-term memory by weakening thalamofrontal synapses, not through interneuron dysfunction.

This study investigates how modulating premotor cortex excitability through deep brain stimulation alleviates motor symptoms in a Parkinson's Disease mouse model by targeting hyperexcitability in M2-STN synapses.

This study shows that aperiodic components of subthalamic local field potentials can predict dopamine depletion in Parkinson's disease and improve machine learning predictions of pathological activity.