Author

Jan‐Hendrik Gosemann

University Hospital LeipzigORCID 0000-0001-9919-0251Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Organism:rat· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Jan RiedeliD· 2 shared Martin LacheriD· 2 shared Nicole Peukert· 2 shared Paula Lieckfeldt· 2 shared Richard KeijzeriD· 2 shared Richard WagneriD· 2 shared Wai Hei TseiD· 2 shared Benedikt Höxter· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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2 shared papersDevelopmental Biology

Jan RiedelORCID

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2 papers

Recent·Most cited

Developmental BiologybioRxivPreprintApr 7, 2023

Overactivated epithelial NF-κB disrupts lung development in human and nitrofen CDH

Florentine Dylong, Jan Riedel, Gaurang M. Amonkar…·Richard Wagner

This study shows that overactivated NF-κB disrupts lung development in CDH and that dexamethasone or curcumenol can rescue lung hypoplasia by normalizing NF-κB signaling.

1 citation

rathuman

𝕏 PostBluesky

Developmental BiologyAnnals of SurgeryPeer-reviewedAug 3, 2022

Proteomic Profiling of Hypoplastic Lungs Suggests an Underlying Inflammatory Response in the Pathogenesis of Abnormal Lung Development in Congenital Diaphragmatic Hernia

Richard Wagner, Paula Lieckfeldt, Hadeesha Piyadasa…·Richard Keijzer

The paper uses proteomic profiling to identify inflammatory processes as a key factor in the pathogenesis of lung hypoplasia in congenital diaphragmatic hernia.

26 citationstop 3%

rathuman

𝕏 PostBluesky