Kevin M. Collins

The paper shows Rho signaling promotes excitability and synaptic transmission in C. elegans egg-laying circuit by acting pre- and postsynaptically.

This study shows hyperosmotic conditions inhibit C. elegans egg laying through sensory and biophysical mechanisms involving HSN neurons and hydrostatic pressure gradients.

This paper shows that C. elegans uv1 neuroendocrine cells directly respond to mechanical activation, using L-type Ca2+ channels, without requiring synaptic or peptidergic signals.

The paper shows that stretch-dependent mechanosensory feedback from vulval muscles regulates egg-laying circuit activity and behavior in C. elegans by modulating Ca2+ transients in both muscles and neurons.

The paper shows that Go signaling in HSN neurons inhibits egg-laying activity and allows stretch-dependent modulation of egg laying in C. elegans.

The paper investigates how serotonin signaling through Gq, PLCβ, and Trio RhoGEF promotes egg-laying behavior in C. elegans by modulating DAG levels and distinct effector pathways.

The paper identifies sex-specific VC neurons in C. elegans that are mechanically activated and facilitate serotonin-induced egg laying by coordinating muscle contractility and mechanosensory feedback.

The paper shows that HSN neurons in C. elegans release both serotonin and NLP-3 to coordinate egg-laying muscle activity, with the two signals acting redundantly.

The paper shows that homeostatic feedback from vulval muscles modulates serotonergic neuron activity in C. elegans to regulate egg-laying behavior.

The paper investigates the neural circuit controlling C. elegans egg-laying behavior, identifying signals that switch between active and inactive states.