Kazuto Kawamura

The paper shows that hil-1/H1-0, a nutrient-regulated linker histone, mediates resilience and restoration during fasting-refeeding in C. elegans and human cells.

This study shows that miR-1 regulates lysosomal v-ATPase and biogenesis in C. elegans muscle to affect contractility during proteotoxic stress and ageing.

This study identifies HDA-3 as a genetic factor contributing to locomotor ability maintenance in C. elegans and finds that mutations in HDA-3 and related genes lead to progressive locomotor decline.

The paper identifies C. elegans mutants with shortened locomotor healthspan, finding that Elongator complex mutations impair adult locomotor function.