Author

Feng Bi

Texas A&M Health Science CenterORCID 0000-0002-0527-5105Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Jun XiaiD· 2 shared Maryam Vaziripour· 2 shared Shiwei Yin· 2 shared Christopher Ian AmosiD· 1 shared Gail F. FernandesiD· 1 shared Jihye Yun· 1 shared Shannon E. SlewitzkeiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

7 top collaborators · 21 mutual co-author links

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2 shared papersPharmacology & Toxicology

Jun XiaORCID

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2 papers

Recent·Most cited

Pharmacology & ToxicologybioRxivPreprintSep 25, 2026

Aquaporin-9 and aquaporin-10 but not aquaporin-3 confer susceptibility to dimethylarsinic acid genotoxicity in human cells

Shiwei Yin, Feng Bi, Maryam Vaziripour, Jun Xia

The paper shows that AQP9 and AQP10 increase DMA(V)-induced DNA damage in human cells, while AQP3 and AQP7 do not, indicating their role in arsenic genotoxicity.

DOI 10.64898/2026.09.20.752519

humanexperimental assay

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Cell BiologybioRxivPreprintSep 24, 2026

Aquaporin-3 amplifies arsenite genotoxicity as a dose-dependent gene-by-environment interaction in human cells

Shiwei Yin, Feng Bi, Maryam Vaziripour…·Jun Xia

This study shows that AQP3 amplifies arsenite-induced DNA damage in a dose-dependent manner, indicating a gene-by-environment interaction that influences genotoxicity.

1 citation

humanbulk RNA-seq

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