Xurde M. Caravia

This study shows that miR-29 is essential for adult cardiomyocyte function, as its deficiency leads to dilated cardiomyopathy and mitochondrial dysfunction in mice and human cells.

This study shows that ATG4D deficiency in mice causes late-onset cardiomyopathy and stress-induced heart failure, and ATG4D repression is linked to human cardiac remodeling.

This study identifies miR-29a-3p as a myokine secreted during resistance exercise that coordinates metabolic responses and miRNA expression in muscle and liver.