Author

Tanmoy Roychowdhury

University of MichiganORCID 0000-0002-1187-1720Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Genetic & Genomic Medicine· 1 Genomics· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 2 Approach:statistical model· 2 Research Focus:GWAS / association mapping· 1 Assay:GWAS summary statistics· 1 Research Focus:variant effect prediction· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Ben Michael BrumptoniD· 2 shared Brooke N. WolfordiD· 2 shared Ida L. SurakkaiD· 2 shared Jifeng ZhangiD· 2 shared Jonas Bille NielseniD· 2 shared Sarah E. GrahamiD· 2 shared Whitney HornsbyiD· 2 shared Y. Eugene CheniD· 2 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersGenetic & Genomic Medicine

Ben Michael BrumptonORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Genetic & Genomic MedicinemedRxivPreprintMay 27, 2022

Multi-ancestry GWAS deciphers genetic architecture of abdominal aortic aneurysm and highlights PCSK9 as a therapeutic target

Tanmoy Roychowdhury, Derek M. Klarin, Michael G. Levin…·Scott M. Damrauer

This study identifies 144 genetic loci associated with abdominal aortic aneurysm and highlights PCSK9 as a therapeutic target through Mendelian Randomization and pre-clinical validation.

9 citations

humanGWAS summary statistics

𝕏 PostBluesky

GenomicsbioRxivPreprintApr 2, 2019

Loss-of-function genomic variants with impact on liver-related blood traits highlight potential therapeutic targets for cardiovascular disease

Jonas Bille Nielsen, Oren Rom, Ida L. Surakka…·Y. Eugene Chen

The paper identifies loss-of-function variants affecting liver-related blood traits that reduce CVD risk without increasing metabolic disease risk, highlighting potential therapeutic targets.

4 citations

humanwhole-genome sequencing

𝕏 PostBluesky