Author

William R. Miller

Houston MethodistORCID 0000-0002-1518-2973Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:bacteria (other)· 1 Research Focus:differential expression· 1 Organism:E. coli· 1 Organism:human· 1 Approach:statistical model· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Cesar A. AriasiD· 3 shared Alex E DeyanoviD· 1 shared An Q DinhiD· 1 shared Ana Paula StrelingiD· 1 shared Andie M DeTranaltesiD· 1 shared Ashleigh MilleriD· 1 shared Blake HansoniD· 1 shared Catherine LiuiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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3 shared papersMicrobiology

Cesar A. AriasORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Loss of the Ferripyochelin Receptor FptA Drives Reduced Cefiderocol Susceptibility and Impairs Fitness in Pseudomonas aeruginosa PA14

Donghoon Kang, Rodrigo P. Baptista, Salvador I. Drusin, Diego Martín Moreno, Cesar A. Arias, William R. Miller

This study identifies FptA inactivation as a key step in cefiderocol resistance in Pseudomonas aeruginosa PA14, revealing resistance mechanisms and fitness costs.

Antimicrobial Agents and Chemotherapy · Feb 12, 2026Peer-reviewedMicrobiology

Clinical and Genomic Characterization of Recalcitrant Enterococcal Bacteremia: A Multicenter Prospective Cohort Study (VENOUS)

Shelby R. Simar, Truc Thanh Tran, Kirsten Rydell, Rachel Atterstrom, Pranoti V. Sahasrabhojane, An Q Dinh, Marissa G Schettino, Haley Slanis, Alex E Deyanov, Andie M DeTranaltes, Dierdre B. Axell‐House, William R. Miller, Jose Manuel Munita, David Tobys, Harald Seifert, , César A. Arias

The paper characterizes recalcitrant enterococcal bacteremia through clinical and genomic analysis, finding species-specific genetic adaptations in persistent infections.

The Journal of Infectious Diseases · Jul 6, 2025Peer-reviewedGenomics

LiaS-dependent activation of the MadR regulon enables cross-talk between Enterococcus faecalis cell envelope defense systems

Kara S. Hood, Samie A Rizvi, Guillermo Hoppe-Elsholz, Ana Paula Streling, Diana Panesso, Shivendra Pratap, Yousif Shamoo, César A. Arias, William R. Miller

This study shows that the MadR regulon can be activated independently of MadS through cross-talk with LiaS, enhancing Enterococcus faecalis resistance to daptomycin.

bioRxiv · Jun 19, 2025 · PreprintMicrobiology