Author

Hayrettin Tumani

Universität UlmORCID 0000-0002-1647-6201Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Approach:experimental assay· 1 Research Focus:GWAS / association mapping· 1 Assay:GWAS summary statistics· 1

Frequent Co-Authors

Abel Viejo‐BorbollaiD· 1 shared Adalbert KrawczykiD· 1 shared Adam SantanielloiD· 1 shared Alberto CataneseiD· 1 shared Alicia GorethiD· 1 shared An GorisiD· 1 shared Angela RosenbohmiD· 1 shared Anna Ponomarenko· 1 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperNeuroscience

Abel Viejo‐BorbollaORCID

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2 papers

Recent·Most cited

Herpes simplex virus infection promotes ALS pathology through ICP0-mediated PML body disruption

Dennis Freisem, Daniel Rombach, Sarah J. Brockmann, Anne Fink, Zoé Engels, Aurora De Luna, Dhiraj Acharya, Helene Hoenigsperger, Alicia Goreth, Sinje Tigges, Isabel Hagmann, Michiel van Gent, Fabian Zech, Anna Ponomarenko, Angela Rosenbohm, , Karin M. Danzer

This study shows HSV infection increases ALS risk and promotes TDP-43 pathology through ICP0-mediated disruption of PML bodies.

bioRxiv · Mar 27, 2026 · PreprintNeuroscience

Low frequency and rare coding variation contributes to multiple sclerosis risk

Mitja Mitrovič, Nikolaos A. Patsopoulos, Ashley Harris Beecham, Theresa Dankowski, An Goris, Bénédicte Dubois, Marie Beatrice D'hooghe, Robin Lemmens, Philip Van Damme, Helle Bach Søndergaard, Finn Thorup Sellebjerg, Per Soelberg Sørensen, Henrik Ullum, Lise Wegner Thørner, Thomas Mears Werge, , Hakon H. Hakonarson

The paper identifies low-frequency coding variants contributing to multiple sclerosis risk using a large case-control study.

Cell · Oct 18, 2018Peer-reviewedGenetics