Sandra L. Schmid

This study shows that VAMP8 depletion impairs clathrin-mediated endocytosis by disrupting endocytic cargo recycling and rerouting it to lysosomal degradation.

This study shows that GAK-Hsc70 early recruitment promotes CCP stabilization and curvature development during clathrin-mediated endocytosis.

This study identifies CCDC32 as an endocytic accessory protein that stabilizes clathrin-coated pits and drives their invagination, with mutations linked to cardio-facio-neuro-developmental syndrome.