Author

Peter Aldiss

University of CopenhagenORCID 0000-0002-1809-2092Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Denise Oró· 1 shared Hayley B. BurmiD· 1 shared Henrik H. HanseniD· 1 shared Leire ToricesiD· 1 shared Malte Hasle NielseniD· 1 shared Marit E. JørgenseniD· 1 shared Matthew Paul GillumiD· 1 shared Mauro D’AmatoiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPhysiology

Denise Oró

Mutual co-authors in cluster

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2 papers

Recent·Most cited

FGF21 deletion mildly exacerbates hepatic dysfunction in MASH diet and alcohol fed rats

Peter Aldiss, Malte Hasle Nielsen, Hayley B. Burm, Denise Oró, Henrik H. Hansen, Michael Feigh, Matthew Paul Gillum

This study shows that FGF21 deletion worsens hepatic dysfunction in rats fed a MASH diet and alcohol, but does not affect body weight or liver transcriptome in the diet model.

npj Metabolic Health and Disease · May 28, 2025Peer-reviewedPhysiology

Sucrase isomaltase dysfunction influences dietary sucrose intake and preference

Peter Aldiss, Leire Torices, Stina Ramne, Marit E. Jørgensen, Sucrase-Isomaltase Working Group, Mauro D’Amato, Mette Korre Andersen

This study shows that SI gene variants affect sucrose intake and preference in mice and humans, linking SI dysfunction to reduced consumption and disliking of sucrose-rich foods.

bioRxiv · Apr 18, 2024 · PreprintGenetics