William F.C. Baaré

This study investigates the relationship between brain atrophy and memory decline in aging using longitudinal data, finding a nonlinear association that intensifies with age and is influenced by APOE ε4 carriership.

This study assesses the reliability of structural brain change in cognitively healthy adults over time, finding that longer follow-up periods significantly improve reliability compared to increasing observation frequency.

The paper investigates the effects of short sleep on brain structure and cognitive function, finding that short sleepers without daytime tiredness have larger brain volumes but slightly lower cognitive scores.

The paper examines longitudinal DTI data to show that neuroticism is associated with cingulum microstructural asymmetry, with stable associations in females and emerging ones in males during adolescence.

The paper investigates how general cognitive ability relates to cortical brain volume changes over time, finding associations with both brain reserve and maintenance mechanisms.

The paper quantifies individual and regional differences in cortical thinning midpoints using longitudinal neuroimaging data and a nonlinear mixed model.

This study examines the relationship between sleep duration and brain structure using MRI data, finding that 6.8 hours of sleep is associated with the most favorable brain outcomes, with genetic analyses suggesting homeostatic regulation of sleep duration.

This study finds that adult brain age is more influenced by early life factors like birth weight and polygenic scores than by longitudinal brain decline.

The paper examines how education and income relate to brain and cognitive differences across European and US cohorts, finding heterogeneous effects and stronger associations in US samples.

This study shows that white matter maturation in the right preSMA correlates with improvements in motor response cancellation, with developmental plateaus around 13-14 years.

This study examines neural responses to repeated sounds in individuals with 22q11.2 deletion syndrome, finding reduced adaptation but intact prediction, linked to negative symptom severity.