Alessandra Angelucci

The paper examines how visual stimulus size modulates neural response variability and shared variability across layers of macaque primary visual cortex, finding layer-specific effects that challenge the idea that surround suppression always reduces variability.

This study investigates how parvalbumin-expressing inhibitory interneurons in primate visual cortex control response gain and orientation-tuning across cortical layers.

This study evaluates the acute biological response to Utah Optrode Array implantation in macaque cortex, identifying design parameters that minimize tissue damage and improve biocompatibility.

The paper validates and characterizes viral vectors for targeting GABAergic and parvalbumin interneurons in marmoset primary visual cortex, showing high specificity and efficiency.

The paper investigates how V2 receptive fields in primates emerge from V1 inputs using anatomical tracing and functional imaging, finding that linear combinations of V1 inputs can explain V2 selectivity and texture sensitivity.

This paper develops and tests an optimized Utah Optrode Array for in vivo optogenetics in non-human primates, showing improved light delivery and spatial selectivity.

The paper introduces an optrode array for precise optogenetic stimulation of deep cortical layers in non-human primates, demonstrating its ability to activate neurons in specific or widespread regions.

This study identifies direct interareal feedback-to-feedforward circuits in primate visual cortex using viral tracing, showing V1 neurons receive monosynaptic feedback from V2.

This study identifies stream-specific feedback connections from V2 to V1 in macaques, showing they mirror feedforward pathways and suggest specialized modulation of V1 responses.

The paper investigates how different layers of primate visual cortex process local and global spatial context through electrophysiological recordings.

This study identifies cellular mechanisms of feedback-mediated modulations in primate visual cortex using optogenetic inactivation and a recurrent network model.