Author

Kyu Sang Joeng

University of PennsylvaniaORCID 0000-0002-1978-0575Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioengineering· 1 Developmental Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:development & morphogenesis· 1 Organism:human· 1 Organism:zebrafish· 1

Frequent Co-Authors

Abbey A. ScottiD· 1 shared Aiden Smith· 1 shared Alistair Duncan CalderiD· 1 shared Alyssa A. Tran· 1 shared Aurélie Clément· 1 shared Bernardo Blanco‐SáncheziD· 1 shared Bon‐Hyeock KooiD· 1 shared Brendan LeeiD· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperGenetics

Abbey A. ScottORCID

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3 papers

Recent·Most cited

mTORC1 and STAT3 signalings are indispensable for in vitro TGFβ1-dependent Three-Dimensional (3D) tendon construct.

Bon‐Hyeock Koo, Aiden Smith, Kyu Sang Joeng

This study shows that mTORC1 and STAT3 signaling are essential for TGFβ1-induced 3D tendon construct formation in vitro.

BMB Reports · May 6, 2025Peer-reviewedBioengineering

Rcn3 is Involved in Postnatal Tendon Development by Regulating Collagen Modification and Fibrillogenesis

Na Rae Park, Snehal S. Shetye, Igor Bogush, Douglas R. Keene, Sara F. Tufa, David M. Hudson, Marilyn Archer, Ling Qin, Louis J. Soslowsky, Nathaniel A. Dyment, Kyu Sang Joeng

This study shows that Rcn3 regulates collagen modification and fibrillogenesis in postnatal tendon development, affecting tendon structure and mechanical properties.

Scientific Reports · May 25, 2021Peer-reviewedDevelopmental Biology

COPB2 haploinsufficiency causes a coatopathy with osteoporosis and developmental delay

Ronit Marom, Lindsay Catherine Burrage, Aurélie Clément, Bernardo Blanco‐Sánchez, Rossella Venditti, Mahim Jain, Ingo Grafe, Daryl A. Scott, Jill Anne Rosenfeld, Vernon Reid Sutton, Marwan S. Shinawi, Ghayda Mirzaa, Catherine J DeVile, Rowenna Roberts, Alistair Duncan Calder, , Brendan Lee

This study identifies COPB2 haploinsufficiency as a cause of a coatopathy with osteoporosis and developmental delay, showing disrupted collagen trafficking and potential treatment with ascorbic acid.

bioRxiv · Sep 14, 2020 · PreprintGenetics