Aurélie Carlier

This paper introduces a cellular automaton model to study osteogenic differentiation and shows endpoint assays have limited ability to distinguish underlying mechanisms.

This study uses a computational model to investigate crosstalk between cadherin-11 and PDGFR pathways in hMSCs, revealing an additional layer of crosstalk involving β-catenin and an ERK inhibitor.

The paper optimizes biomaterial surface topographies using evolutionary algorithms inspired by natural selection to enhance osteogenic marker expression.

This study investigates how cell shape affects the phenotypic response of human mesenchymal stem cells and identifies molecular mechanisms linking shape to function.