Author
Marcus Ghosh
University College LondonORCID 0000-0002-2428-4605Unclaimed ORCID profile · follow to get notified of new preprints
Biology Disciplines
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2020-2025 · peak 3/yrCo-Author Collaboration Network
10 top collaborators · 21 mutual co-author links7 papers
Recent·Most cited
- NeuroscienceeNeuroPeer-reviewed
Spiking neural network models of sound localisation via a massively collaborative process
Marcus Ghosh, Karim G. Habashy, Francesco De Santis…·Dan F. M. Goodman
The paper describes a massively collaborative project using spiking neural networks to study sound localization, involving 31 researchers across multiple countries.
6 citations - NeurosciencePLoS Computational BiologyPeer-reviewed
Fusing multisensory signals across channels and time
Swathi Anil, Dan F. M. Goodman, Marcus Ghosh
The paper introduces a novel multisensory task to evaluate how well algorithms can integrate signals across sensory channels and time, showing that simple temporal integration can match complex recurrent models.
- NeurosciencePLoS Computational BiologyPeer-reviewed
Nonlinear fusion is optimal for a wide class of multisensory tasks
Marcus Ghosh, Gabriel Béna, Volker Bormuth, Dan F. M. Goodman
The paper demonstrates that nonlinear fusion outperforms linear fusion in multisensory integration tasks when sensory signals are statistically dependent, and shows that spiking neural networks can approximate this nonlinear algorithm.
8 citations - GeneticseLifePeer-reviewed
A simple and effective F0 knockout method for rapid screening of behaviour and other complex phenotypes
François Kroll, Gareth T. Powell, Marcus Ghosh…·Jason Rihel
This paper introduces a CRISPR-Cas9 method for rapid generation of F0 biallelic knockouts in zebrafish, enabling quick screening of complex phenotypes.
349 citationstop 1% - Developmental BiologyScientific ReportsPeer-reviewed
Diverse Species-Specific Phenotypic Consequences of Loss of Function Sorting Nexin 14 Mutations
Dale Bryant, Marian Seda, Emma Peskett…·Philip Stanier
This study examines the phenotypic effects of SNX14 loss-of-function mutations in mice and zebrafish, revealing species-specific consequences despite conserved biochemical roles.