Author

Chiara Parodi

University of MilanORCID 0000-0002-2689-2049Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:Drosophila· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

Cristina GervasiniiD· 2 shared Elisabetta Di FedeiD· 2 shared Paolo GrazioliiD· 2 shared Thomas VaccariiD· 2 shared Valentina MassaiD· 2 shared Aı́da ZuluetaiD· 1 shared Angelo SelicorniiD· 1 shared Anna CeredaiD· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersGenetics

Cristina GervasiniORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

NeuroscienceJournal of Cachexia Sarcopenia and MusclePeer-reviewedMar 10, 2023

Transcriptomic analysis of aging mouse sciatic nerve reveals early pathways leading to sarcopenia

Nicole T. Comfort, Meethila Gade, Madeleine D. Strait…·Diane Bérangère Ré

This study identifies early molecular changes in aging mouse sciatic nerve that precede sarcopenia onset, revealing pathways linked to circadian rhythm and metabolic processes.

17 citations

mousebulk RNA-seq

𝕏 PostBluesky

GeneticsbioRxivPreprintJul 15, 2020

Lithium ameliorates Cornelia de Lange syndrome associated phenotypes in experimental models

Paolo Grazioli, Chiara Parodi, Milena Mariani…·Valentina Massa

This study investigates the effects of lithium on CdLS phenotypes in experimental models, showing rescue of brain and behavioral abnormalities through WNT pathway activation.

DOI 10.1101/2020.07.15.204628

Drosophilamouse

𝕏 PostBluesky

GeneticsbioRxivPreprintMar 31, 2020

Exogenous and endogenous HDAC inhibitor effects in Rubinstein-Taybi syndrome models

Elisabetta Di Fede, Emerenziana Ottaviano, Paolo Grazioli…·Cristina Gervasini

This study investigates the effects of HDAC inhibitors in RSTS models, showing molecular rescue and microbiota changes, and highlights the role of gut microbiota in chromatinopathy.

1 citation

Drosophilaproteomics

𝕏 PostBluesky