Author

Bruno Crestani

Assistance Publique – Hôpitaux de ParisORCID 0000-0002-2961-3455Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Naftali KaminskiiD· 2 shared A. JustetiD· 1 shared Adam SzmuliD· 1 shared Antje PrasseiD· 1 shared Apostolos GalarisiD· 1 shared Argyrios TzouvelekisiD· 1 shared Aurélie CazesiD· 1 shared Bart M. VanaudenaerdeiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

Naftali KaminskiORCID

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2 papers

Recent·Most cited

Cell BiologyScience AdvancesPeer-reviewedMay 8, 2026

The pleuroparenchymal fibroelastosis atlas reveals aberrant cell states and their zonation as an alternate roadmap to lung fibrosis

Jannik Ruwisch, Aurélie Cazes, Lena Maureen Leiber…·Jonas C. Schupp

This paper presents the first single-nucleus RNA sequencing atlas of pleuroparenchymal fibroelastosis, identifying novel cell states and their spatial organization in lung fibrosis.

5 citationstop 1%

humanscRNA-seq

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Molecular BiologybioRxivPreprintJan 25, 2023

SRC-mediated and TKS5-enabled podosome formation is an inherent property of IPF fibroblasts, promoting ECM invasion and pulmonary fibrosis

Ilianna Barbayianni, Paraskevi Kanellopoulou, Dionysiοs Fanidis…·Vassilis Aidinis

This study identifies TKS5-mediated podosome formation in IPF fibroblasts as a driver of ECM invasion and pulmonary fibrosis, with potential therapeutic targeting via src kinase inhibition.

DOI 10.1101/2023.01.25.522705

mousehuman

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