Author

Edmarie Guzmán‐Vélez

Harvard UniversityORCID 0000-0002-3076-0727Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 1 Organism:human· 1 Assay:imaging· 1 Approach:statistical model· 1

Frequent Co-Authors

Ana Y. BaenaiD· 1 shared Clara Vila‐CastelariD· 1 shared Dorothée SchoemakeriD· 1 shared Enmanuelle Pardilla‐DelgadoiD· 1 shared Francisco Javier LoperaiD· 1 shared Ibai Palacio DieziD· 1 shared Jorge SepulcreiD· 1 shared Joshua T. Fox‐FulleriD· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Ana Y. BaenaORCID

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1 paper

Recent·Most cited

Amyloid-β and tau pathologies relate to distinctive brain dysconnectomics in preclinical autosomal-dominant Alzheimer's disease

Edmarie Guzmán‐Vélez, Ibai Palacio Diez, Dorothée Schoemaker, Enmanuelle Pardilla‐Delgado, Clara Vila‐Castelar, Joshua T. Fox‐Fuller, Ana Y. Baena, Reisa Anne Sperling, Keith A. Johnson, Francisco Javier Lopera, Jorge Sepulcre, Yakeel T. Quiroz

This study examines how amyloid-β and tau pathologies affect brain functional connectivity in preclinical Alzheimer's disease carriers, finding distinct dysconnectivity patterns in specific brain regions.

Proceedings of the National Academy of Sciences · Apr 5, 2022Peer-reviewedNeuroscience