Author

Yakeel T. Quiroz

Harvard UniversityORCID 0000-0001-9714-8244Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 4 Research Focus:differential expression· 3 Approach:statistical model· 2 Approach:experimental assay· 1 Assay:imaging· 1 Approach:machine learning (classical)· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

Francisco Javier LoperaiD· 3 shared Ana Y. BaenaiD· 2 shared David Fernando Aguillón NiñoiD· 2 shared Joseph F. Arboleda-VelasqueziD· 2 shared Adam R. FergusoniD· 1 shared Andrés VillegasiD· 1 shared Clara Vila‐CastelariD· 1 shared Daniel Camilo Aguirre-AcevedoiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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Curved chords = mutual co-authorship

3 shared papersNeuroscience

Francisco Javier LoperaORCID

Mutual co-authors in cluster

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4 papers

Recent·Most cited

NeurosciencebioRxivPreprintNov 25, 2025

Serum Lipidome as an Early Peripheral Indicator in Familial Alzheimers Disease

Gloria Patricia Cardona‐Gómez, Ivan Daniel Salomon-Cruz, Laura Alejandra Lozano-Trujillo…·Estela Área-Gómez

The paper identifies a serum lipidomic signature in asymptomatic Alzheimer's disease mutation carriers that differentiates them from non-carriers and correlates with dementia risk factors.

DOI 10.1101/2025.11.25.690392

humanmetabolomics

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NeurosciencebioRxivPreprintMar 28, 2024

Plasma Proteomics of Genetic Brain Arteriosclerosis and Dementia Syndrome Identifies Signatures of Fibrosis, Angiogenesis, and Metabolic Alterations

Jonah N. Keller, Hannah Radabaugh, Nikolaos Karvelas…·Fanny M. Elahi

The paper identifies plasma proteomic signatures associated with CADASIL stages, revealing fibrosis, angiogenesis, and metabolic alterations.

DOI 10.1101/2024.03.28.587249

humanproteomics

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NeurosciencebioRxivPreprintJan 11, 2023

APOE3 Christchurch modulates tau phosphorylation and regulates β-catenin/Wnt/Cadherin signaling in induced pluripotent stem cell-derived cerebral organoids from Alzheimer's cases

Randall C. Mazzarino, Paula Andrea Perez-Corredor, Timothy E. Vanderleest…·Joseph F. Arboleda‐Velásquez

This study shows that the APOE3Ch variant reduces tau phosphorylation and regulates Wnt/Cadherin signaling in cerebral organoids from Alzheimer's cases, suggesting a protective mechanism against tauopathy.

7 citations

humanexperimental assay

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NeuroscienceProceedings of the National Academy of SciencesPeer-reviewedApr 5, 2022

Amyloid-β and tau pathologies relate to distinctive brain dysconnectomics in preclinical autosomal-dominant Alzheimer's disease

Edmarie Guzmán‐Vélez, Ibai Palacio Diez, Dorothée Schoemaker…·Yakeel T. Quiroz

This study examines how amyloid-β and tau pathologies affect brain functional connectivity in preclinical Alzheimer's disease carriers, finding distinct dysconnectivity patterns in specific brain regions.

92 citationstop 1%

humanimaging

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