Author

Makoto Kondo

Osaka Metropolitan UniversityORCID 0000-0002-3220-9859Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Ami Konishi· 1 shared Geneviève KonopkaiD· 1 shared Hideo MatsuzakiiD· 1 shared Noriyoshi UsuiiD· 1 shared Shoichi ShimadaiD· 1 shared Stefano BertoiD· 1 shared Tomoaki NakazonoiD· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 15 mutual co-author links

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1 shared paperNeuroscience

Ami Konishi

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2 papers

Recent·Most cited

NeuroscienceBehavioural Brain ResearchPeer-reviewedOct 27, 2025

Deletion of the 5-HT3A receptor reduces behavioral persistence and enhances flexibility

Tomoaki Nakazono, Makoto Kondo

This study examines the role of the 5-HT3A receptor in behavioral persistence and flexibility by comparing wild-type and knockout mice in operant conditioning tasks.

1 citation

mouseexperimental assay

𝕏 PostBluesky

NeuroscienceTranslational PsychiatryPeer-reviewedApr 24, 2021

Zbtb16 regulates social cognitive behaviors and neocortical development.

Noriyoshi Usui, Stefano Berto, Ami Konishi…·Shoichi Shimada

This study shows that Zbtb16 deletion in mice causes ASD and SCZ-like behaviors and neocortical developmental defects, with associated transcriptomic changes.

56 citationstop 4%

mousestatistical model

𝕏 PostBluesky