Isabel M. Palacios

This study shows that kinesin-1 and kinesin-3 mitigate Aβ42Arc toxicity in Drosophila neurons and rescue behavioral defects.

This study investigates how integrin loss and tissue mechanics disrupt simple epithelial architecture in Drosophila, revealing mechanisms of epithelial breakdown and their relevance to disease.

The paper demonstrates apical compressive forces during neuroepithelial organoid development using artificial cells that mimic cadherin-mediated interactions.

This study identifies integrin-dependent mechanisms essential for maintaining simple epithelial architecture in Drosophila by integrating live imaging, biophysical measurements, and BM property manipulation.