Author

Vidya Rajasekaran

Edinburgh Cancer ResearchORCID 0000-0002-3501-9321Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetic & Genomic Medicine· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:GWAS / association mapping· 1

Frequent Co-Authors

Bradley T. HarrisiD· 2 shared Farhat V N DiniD· 2 shared Kevin DonnellyiD· 2 shared Kevin MyantiD· 2 shared Malcolm G. DunlopiD· 2 shared Marion WalkeriD· 2 shared Morven Allan· 2 shared Susan Mary FarringtoniD· 2 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Bradley T. HarrisORCID

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2 papers

Recent·Most cited

Genetic & Genomic MedicinemedRxivPreprintJul 14, 2026

Genetic Variation at 19q13.33 confers colorectal cancer risk through the interaction of mucosal expression of FUT2 and plasma vitamin B12 levels

Morven Allan, Vidya Rajasekaran, Xue Li…·Malcolm G. Dunlop

DOI 10.64898/2026.07.10.26357741

mousebulk RNA-seq

𝕏 PostBluesky

GeneticsGutPeer-reviewedNov 28, 2024

Genetic variation at 11q23.1 confers colorectal cancer risk by dysregulation of colonic tuft cell transcriptional activator POU2AF2

Vidya Rajasekaran, Bradley T. Harris, Ruby Tamara Osborn…·Susan Mary Farrington

This study identifies rs3087967 as the causal variant at 11q23.1 that increases colorectal cancer risk by dysregulating POU2AF2, a key activator of tuft cell transcription.

8 citations

𝕏 PostBluesky