Author

Ruby Tamara Osborn

Edinburgh Cancer ResearchORCID 0000-0002-3222-1743Unclaimed ORCID profile · follow to get notified of new preprints

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Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1 Assay:scATAC-seq· 1

Frequent Co-Authors

Alison M. MeynertiD· 1 shared Bradley T. HarrisiD· 1 shared Ceres Fernández–RozadillaiD· 1 shared Claire L. SmillieiD· 1 shared Edward Esiri-BloomiD· 1 shared Farhat VN DiniD· 1 shared Graeme R. GrimesiD· 1 shared Ian P.M. TomlinsoniD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperGenetics

Alison M. MeynertORCID

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1 paper

Recent·Most cited

GeneticsGutPeer-reviewedNov 28, 2024

Genetic variation at 11q23.1 confers colorectal cancer risk by dysregulation of colonic tuft cell transcriptional activator POU2AF2

Vidya Rajasekaran, Bradley T. Harris, Ruby Tamara Osborn…·Susan Mary Farrington

This study identifies rs3087967 as the causal variant at 11q23.1 that increases colorectal cancer risk by dysregulating POU2AF2, a key activator of tuft cell transcription.

8 citations

humanmouse

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