Author

Erin M Ritchie

University of California San DiegoORCID 0000-0002-3558-3029Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Binhai ZhengiD· 2 shared Yishi JiniD· 2 shared Yunbo LiiD· 2 shared Cai Qi· 1 shared Christopher L SteinkeiD· 1 shared Dilan AcariD· 1 shared Lizhen CheniD· 1 shared Qianyi PuiD· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 27 mutual co-author links

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2 shared papersNeuroscience

Binhai ZhengORCID

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2 papers

Recent·Most cited

Translatome analysis reveals cellular network in DLK-dependent hippocampal glutamatergic neuron degeneration

Erin M Ritchie, Dilan Acar, Siming Zhong, Qianyi Pu, Yunbo Li, Binhai Zheng, Yishi Jin

The paper identifies DLK-dependent translatome changes in hippocampal glutamatergic neurons, revealing cell-type-specific molecular signatures and potential roles in neurodegeneration.

bioRxiv · Jul 10, 2024Neuroscience

Activation of MAP3K DLK and LZK in Purkinje Cells Causes Rapid and Slow Degeneration Depending on Signaling Strength

Yunbo Li, Erin M Ritchie, Christopher L Steinke, Cai Qi, Lizhen Chen, Binhai Zheng, Yishi Jin

This study shows that activating DLK or LZK in Purkinje cells leads to rapid or slow degeneration, respectively, with LZK's effect modulated by CELF2 and Mkk7 splicing.

eLife · Jan 21, 2021Peer-reviewedNeuroscience