Yishi Jin

The paper investigates the role of lipid metabolism in axon maintenance and regeneration in C. elegans, showing that DIP-2 counterbalances phospholipid synthesis to prevent age-related axonal defects.

The paper identifies a network involving DIP-2, SAX-2, PAD-1, and TAT-5 that regulates neuronal morphology by controlling extracellular vesicle release in C. elegans.

The paper identifies DLK-dependent translatome changes in hippocampal glutamatergic neurons, revealing cell-type-specific molecular signatures and potential roles in neurodegeneration.

The paper investigates the effects of PACS1 and PACS2 variants on protein localization in C. elegans, showing altered localization and functional conservation with human proteins.

The paper identifies LIN-66 as a mediator of eIF-3.G-dependent protein translation in C. elegans motor neurons, revealing a cold-shock domain critical for its function.

This study identifies a role for EIF-3.G in regulating neuronal activity through mRNA translation efficiency in C. elegans.

This study investigates the tissue-specific functions of C. elegans JPH-1, showing its role in neurotransmission and muscle contraction, and its interaction with calcium channels.

This study shows that activating DLK or LZK in Purkinje cells leads to rapid or slow degeneration, respectively, with LZK's effect modulated by CELF2 and Mkk7 splicing.

This study shows that ZDHHC17 couples DLK and NMNAT2 pathways to control both somal and distal axon degeneration after optic nerve crush.

The paper identifies up-regulated neuropeptides in C. elegans with altered neuronal activity, showing their role in modulating motor circuit function and behavior.

The paper identifies class A motor neurons in C. elegans as intrinsic oscillators driving reverse locomotion without premotor interneurons.