Mayssa H. Mokalled

This study identifies elevated phagocytic capacity as essential for innate spinal cord repair by comparing zebrafish and mammalian immune responses.

This study identifies Hapln1-HA signaling as a pro-regenerative mechanism in zebrafish spinal cord injury by enhancing progenitor cell proliferation and repair.

This study identifies sox2+ progenitors as critical for zebrafish spinal cord regeneration and shows Bach1 transcription factors regulate their activation and quiescence.

The paper examines whether spinal cord regeneration in adult zebrafish recapitulates embryonic development by analyzing single-cell RNA-seq data across development, homeostasis, and regeneration.

The paper shows that systemic muscle injury in zebrafish activates Hb-EGF in the epidermis, which is necessary for systemic repair, while local injury does not require Hb-EGF.

This study uses single nuclear RNA sequencing to analyze spinal cord regeneration in zebrafish, identifying neurogenesis and plasticity as key mechanisms for recovery.

The paper tracks functional recovery in zebrafish after spinal cord injury and identifies gait quality as a predictor of regeneration outcomes.

This study identifies the role of progenitor-derived glia in zebrafish spinal cord regeneration through genetic and molecular analyses.

This study shows myostatin b inhibits neurogenesis in zebrafish spinal cord, and its loss improves functional recovery without affecting axonal or glial bridging.

This study develops an efficient CRISPR/Cas9 pipeline for identifying genes involved in spinal cord repair in adult zebrafish through mutagenesis and behavioral screening.

The paper identifies Yap-Ctgf signaling as a driver of glial bridging and spinal cord regeneration in zebrafish through EMT activation in ependymal progenitors.