Author

Shamila D. Alipoor

Linköping UniversityORCID 0000-0002-3855-2682Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Lovisa Ellinger· 1 shared Maria LermiD· 1 shared Mehdi Seyed MirsaeidiiD· 1 shared Sadaf KalsumiD· 1 shared Shumaila SayyabiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

5 top collaborators · 6 mutual co-author links

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1 shared paperMolecular Biology

Lovisa Ellinger

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2 papers

Recent·Most cited

Molecular BiologybioRxivPreprintJan 12, 2026

Epigenetic Crosstalk Between BCG-Infected Macrophages and Naive Monocytes Potentiates Antimycobacterial Activity

Lovisa Ellinger, Shamila D. Alipoor, Shumaila Sayyab…·Maria Lerm

This study shows that BCG-infected macrophages release EVs that epigenetically reprogram naive monocytes, enhancing their antimycobacterial activity against Mtb.

DOI 10.64898/2026.01.12.698601

humanbulk RNA-seq

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GeneticsCellsPeer-reviewedAug 9, 2021

Inborn errors E778K and G908R in NOD2 gene increase risk of nontuberculous mycobacterial infection: a computational study

Shamila D. Alipoor, Mehdi Seyed Mirsaeidi

This study identifies two NOD2 gene variants, E778K and G908R, as increasing susceptibility to nontuberculous mycobacterial infections through computational analysis of their structural and functional impacts.

14 citations

humanstatistical model

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