Author

David J. Salant

Boston UniversityORCID 0000-0002-3866-3120Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Assay:ATAC-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Alexander Rubin· 1 shared Amar J. MajmundariD· 1 shared Andrei PozniakovskyiD· 1 shared Anthony A. HymaniD· 1 shared Arathi Ranga· 1 shared Cijiang HeiD· 1 shared David A. BalliD· 1 shared Fatima Danish Sheikh· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCell Biology

Alexander Rubin

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2 papers

Recent·Most cited

PathologybioRxivPreprintNov 10, 2025

Fosl2 regulates the transition from parietal epithelial cells to myofibroblasts in the kidney

Robert D. Bronstein, Monica Patricia Revelo, Fatima Danish Sheikh…·Sandeep K. Mallipattu

This study identifies Fosl2 as a regulator of PEC-to-myofibroblast transition in kidney disease using mouse models and human biopsies.

1 citation

𝕏 PostBluesky

Cell BiologybioRxivPreprintJan 2, 2025

Mesoscale landscaping of the TRIM protein family reveals a novel human condensatopathy

Hari Singh, Vineeta Sharma, Jik Nijssen…·Amar J. Majmundar

This study investigates mesoscale organization of TRIM proteins, finding that TRIM8 truncations cause condensatopathy by disrupting condensation and signaling pathways.

1 citation

humanproteomics

𝕏 PostBluesky