Author

C.-S. Park

Georgia Institute of TechnologyORCID 0000-0002-3975-1202Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

C.-s. Park4 papers

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Dong Won KangiD· 2 shared Eun Ju SongiD· 2 shared Hanjoong JoiD· 2 shared Jay Aaron Bowman-KiriginiD· 2 shared Kyung In BaekiD· 2 shared Leandro ChoiiD· 2 shared Ruei-Chun HungiD· 2 shared Yerin KimiD· 2 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersPhysiology

Dong Won KangORCID

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2 papers

Recent·Most cited

Flow-sensitive HEG1 controls eNOS activity to prevent endothelial dysfunction, hypertension, and atherosclerosis

Michael D. Clark, Yerin Kim, César A. Romero, Dong Won Kang, Kyung In Baek, Eun Ju Song, Cailin E. Kellum, Jay Aaron Bowman-Kirigin, C.-S. Park, Ruei-Chun Hung, Leandro Choi, Vir Kapoor, Shoutaro Tsuji, Jennifer S. Pollock, Hanjoong Jo

This study identifies HEG1 as a flow-sensitive protein that regulates eNOS activity, preventing endothelial dysfunction, hypertension, and atherosclerosis.

Arteriosclerosis Thrombosis and Vascular Biology · Oct 9, 2025Peer-reviewedPhysiology

Disturbed Flow Induces Reprogramming of Endothelial Cells to Immune-like and Foam Cells under Hypercholesterolemia during Atherogenesis

C.-S. Park, Kyung In Baek, Ruei-Chun Hung, Leandro Choi, Kiyoung Jeong, Paul Kim, Andrew Keunho Jahng, Jung‐Hyun Kim, Mostafa Meselhe, Ashwin Kannan, Chien-Ling Chou, Dong Won Kang, Eun Ju Song, Yerin Kim, Jay Aaron Bowman-Kirigin, , Hanjoong Jo

This study validates the two-hit hypothesis showing disturbed flow and hypercholesterolemia together reprogram endothelial cells to immune-like and foam cells during atherogenesis.

bioRxiv · Mar 6, 2025 · PreprintCell Biology