Jarkko Salojärvi

This paper investigates how whole-genome duplication affects biosynthetic gene clusters in Strychnos species, showing that pathway function can persist despite fragmentation and regulatory rewiring.

This study uses single-cell and spatial transcriptomics to identify B chromosome contributions to invasiveness in Phragmites australis.

This study analyzes 200 woodland strawberry genomes to reveal population structure and demographic history across glacial-interglacial cycles, identifying genetic clusters and refugia.

This study investigates genomic mechanisms of coexistence in a tropical flora, finding stabilising selection and ecological trade-offs between defence and growth.

This study investigates the evolutionary history of BBX transcription factors across green plants, identifying structural groups, domain dynamics, and lineage-specific innovations.

This paper improves population genetics statistics by incorporating basecalling uncertainties and reducing median bias in estimators.

The paper investigates how centromere divergence and allopolyploidy influence genome evolution in carnivorous sundews, revealing structural differences between species with distinct chromosomal architectures.

This study identifies conserved meiotic gene expression patterns in autopolyploid plants across angiosperms, supporting a shared evolutionary response to whole genome duplication.

The paper reconstructs the evolutionary history of Betula pubescens, showing it originated from an autoploid event and later hybridized with multiple species, with introgressed alleles contributing to climate adaptation and meiotic stability.

The paper analyzes the genome and population genomics of Coffea arabica to reveal its diversification history and breeding-related genetic changes.

This paper presents the avocado genome, revealing its phylogenetic position, introgressive hybridization, and pathogen-driven gene space adaptation.

The paper shows RCD1 interacts with ANAC transcription factors to regulate chloroplastic and mitochondrial retrograde signaling via ROS-dependent mechanisms.