Author

Tsze Yin Tan

University of WashingtonORCID 0000-0002-4119-736XUnclaimed ORCID profile · follow to get notified of new preprints

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Organism:C. elegans· 1 Research Focus:differential expression· 1 Assay:metabolomics· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Amaury Cazenave‐GassiotiD· 1 shared Barry B. HalliwelliD· 1 shared Diogo BarardoiD· 1 shared Emelyne TeoiD· 1 shared Hyung-Seok KimiD· 1 shared Jan GruberiD· 1 shared Jean‐Paul KovalikiD· 1 shared Jianhong ChingiD· 1 shared

Activity by Year

2019-2019 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Amaury Cazenave‐GassiotORCID

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1 paper

Recent·Most cited

Metabolic stress is a primary pathogenic event in transgenic Caenorhabditis elegans expressing neuronal human amyloid-β

Emelyne Teo, Sudharshan Ravi, Diogo Barardo, Hyung-Seok Kim, Sheng Fong, Amaury Cazenave‐Gassiot, Tsze Yin Tan, Jianhong Ching, Jean‐Paul Kovalik, Markus Rene Wenk, Rudiyanto Gunawan, Philip Keith Moore, Barry B. Halliwell, Nicholas S. Tolwinski, Jan Gruber

This study identifies metabolic dysfunction, particularly in the TCA cycle, as an early and causative event in Alzheimer's disease using a transgenic C. elegans model.

eLife · Oct 14, 2019Peer-reviewedNeuroscience