Madison R. Tetzlaff

The paper identifies sex-dependent roles of paraventricular thalamus neurons in mediating the effects of early-life adversity on adult reward behaviors in mice.

The paper identifies disrupted CRH signaling as a cause of reward deficits after early-life stress and shows CRHR2 activation can reverse anhedonia-like behaviors.

The paper investigates how early-life adversity affects reward behaviors through peptide signaling in the paraventricular thalamus and identifies CRHR1 as a molecular target for intervention.

The paper defines a histology-informed MRI mask for the human paraventricular nucleus of the thalamus, showing spatial overlap with existing masks but with additional posterior voxels and consistent functional connectivity patterns.

The paper identifies distinct fastigial output channels in mice and shows that stimulating neurons targeting the central lateral thalamus, but not other regions, effectively inhibits hippocampal seizures.

The paper identifies a novel hippocampal neuronal population, LINCs, which are GABAergic interneurons with long-range axons, influencing hippocampal and extrahippocampal network function.