Author

Alison Mao

University of TorontoORCID 0000-0002-4428-6077Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Organism:human· 1 Organism:mouse· 1 Assay:protein structure· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Erica StuartiD· 2 shared Joel C. WattsiD· 2 shared Le yao LiiD· 2 shared Nicholas R. G. SilveriD· 2 shared Raphaella W. L. SoiD· 2 shared Benedikt FriegiD· 1 shared Bradley T. HymaniD· 1 shared Christopher Situ· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersNeuroscience

Erica StuartORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintJun 10, 2026

α-Synuclein strain homogeneity in multiple system atrophy clinical subtypes

Heather H. C. Lau, Nicholas R. G. Silver, Surabhi Mehra…·Joel C. Watts

This study investigates whether distinct α-synuclein strains contribute to clinical subtypes of multiple system atrophy (MSA) and finds no differences in strain properties between MSA-C and MSA-P.

DOI 10.64898/2026.06.05.730469

humanproteomics

𝕏 PostBluesky

NeuroscienceNeuronPeer-reviewedFeb 27, 2026

Stochastic Misfolding Drives the Emergence of Distinct α-Synuclein Strains

Raphaella W. L. So, Benedikt Frieg, José D. Camino…·Joel C. Watts

This study shows that stochastic misfolding leads to the formation of distinct α-synuclein strains both in vitro and in vivo, resulting in different disease phenotypes.

9 citationstop 1%

mouseprotein structure

𝕏 PostBluesky