Author

Svetlana Vakkilainen

Karolinska University HospitalORCID 0000-0002-4573-3297Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Svetlana Vakkilainen2 papers · Childrens Hospital and Pediatric Research Center, University of Helsinki and Helsinki University Hospital; Faculty of Medicine, University of Helsinki, Helsinki, Finland;

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:CRISPR screen· 1 Approach:deep learning· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Anniken Solveig Matheson Sollano· 1 shared Arne LindqvistiD· 1 shared Børre FevangiD· 1 shared Camilla Schalin-Jäntti· 1 shared Eira LeinoneniD· 1 shared Eliisa KekäläineniD· 1 shared Emma Maria HaapaniemiiD· 1 shared Fulya TaylaniD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperMolecular Biology

Anniken Solveig Matheson Sollano

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2 papers

Recent·Most cited

Molecular BiologyFrontiers in Genome EditingPeer-reviewedSep 30, 2026

Protocol for Non-viral HDR-based CRISPR/Cas9 platform for small custom editing in primary T cells

Katariina Mamia, Anniken Solveig Matheson Sollano, Vanja Lundberg…·Emma Maria Haapaniemi

This paper presents a non-viral HDR-based CRISPR/Cas9 protocol for editing primary T cells with up to 80% HDR efficiency, targeting small pathogenic variants in patient and healthy donor cells.

DOI 10.3389/fgeed.2026.1817322

humanCRISPR screen

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Cell BiologyEMBO ReportsPeer-reviewedMay 16, 2026

Cumulative microscopy reveals cellular states in fibroblasts from patients with genetic disorders

Karen Akopyan, Zhiyu Hao, Susann Karlberg…·Arne Lindqvist

The paper introduces cumulative microscopy to analyze cellular states in fibroblasts from patients with genetic disorders, finding replication stress in CHH but not MUL.

DOI 10.1038/s44319-026-00859-5

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