Author

Romain Rey

Centre National de la Recherche ScientifiqueORCID 0000-0002-4603-3575Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Genetics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:gene regulatory network inference· 1 Research Focus:variant effect prediction· 1

Frequent Co-Authors

Caroline DemilyiD· 1 shared Elliott ReesiD· 1 shared George KiroviD· 1 shared Hai‐Gwo HwuiD· 1 shared Hugo CreethiD· 1 shared James WaltersiD· 1 shared Marion LeboyeriD· 1 shared Michael C. O’DonovaniD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperNeuroscience

Caroline DemilyORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Dissociation Between Genetic Risk and Transcriptional Output in Schizophrenia: A Cross-Tissue Meta-Analysis of CSMD1 and CSMD2 Expression

Mohamed-ElMehdi Boughanmi, Marion Leboyer, Caroline Demily, Romain Rey

The paper finds that CSMD2 is overexpressed in brain tissues of schizophrenia patients, while CSMD1 shows no difference, suggesting a dissociation between genetic risk and transcriptional output.

bioRxiv · Mar 20, 2026 · PreprintNeuroscience

Schizophrenia, autism spectrum disorders and developmental disorders share specific disruptive coding mutations

Elliott Rees, Hugo Creeth, Hai‐Gwo Hwu, Wei J. Chen, Ming T. Tsuang, Stephen J. Glatt, Romain Rey, George Kirov, James Walters, Peter Holmans, Michael John Owen, Michael C. O’Donovan

The paper identifies shared disruptive coding mutations between schizophrenia, autism spectrum disorders, and other developmental disorders, suggesting overlapping molecular aetiology.

Nature Communications · Sep 9, 2021Peer-reviewedGenetics