Author

Paweł Śledziński

Institute of Bioorganic Chemistry, Polish Academy of SciencesORCID 0000-0002-4671-5189Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1

Signature Assays, Organisms & Methods

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Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Research Focus:protein structure prediction· 1 Research Focus:variant calling· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Marianna Smielowska· 1 shared Marta OlejniczakiD· 1 shared Mateusz NowaczykiD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

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3 top collaborators · 3 mutual co-author links

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1 shared paperMolecular Biology

Marianna Smielowska

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1 paper

Recent·Most cited

Molecular BiologyBMC BiologyPeer-reviewedDec 3, 2024

CRISPR/Cas9-induced double-strand breaks in huntingtin locus lead to CAG repeat contraction through the extensive DNA end resection and homology-mediated repair

Paweł Śledziński, Mateusz Nowaczyk, Marianna Smielowska, Marta Olejniczak

This study shows that CRISPR/Cas9-induced DSBs in the huntingtin locus cause CAG repeat contraction through DNA end resection and homology-mediated repair in human cells.

4 citations

humanDNA methylation

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