Author

Mohamed H. Farah

Johns Hopkins UniversityORCID 0000-0002-4745-8030Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Neuroscience· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 1 Organism:mouse· 1

Frequent Co-Authors

Balaji G. Vijayakumar· 1 shared Emily G. ThompsoniD· 1 shared Erica R. Kent· 1 shared Jeffrey D. RothsteiniD· 1 shared Olivia SpeadiD· 1 shared Sean MilleriD· 1 shared Svetlana Vidensky· 1 shared Thomas PhilipsiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperNeuroscience

Balaji G. Vijayakumar

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Astrocyte MCT1 expression does not contribute to the axonal degenerative phenotype observed with ubiquitous MCT1 depletion.

Thomas Philips, Emily G. Thompson, Olivia Spead, Balaji G. Vijayakumar, Erica R. Kent, Sean Miller, Treasure Nwokeleme, Svetlana Vidensky, Mohamed H. Farah, Jeffrey D. Rothstein

This study shows that astrocyte MCT1 depletion does not cause axonal degeneration, unlike oligodendrocyte MCT1 depletion, suggesting astrocyte MCT1 is not essential for axonal energy homeostasis with aging.

Glia · Dec 2, 2025Peer-reviewedNeuroscience