Author

Emily G. Thompson

Johns Hopkins UniversityORCID 0000-0002-5615-2893Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 3 Approach:experimental assay· 2 Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Balaji G. Vijayakumar· 2 shared Erica R. Kent· 2 shared Jeffrey D. RothsteiniD· 2 shared Olivia SpeadiD· 2 shared Thomas PhilipsiD· 2 shared Alec M. Wright· 1 shared Alex J. CallahaniD· 1 shared Anindita MitraiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersNeuroscience

Balaji G. Vijayakumar

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3 papers

Recent·Most cited

BioengineeringbioRxivPreprintJun 11, 2026

High-throughput discovery of arginine-depleted peptides enables effective antisense delivery for Duchenne muscular dystrophy

Charlotte E. Farquhar, Nathan W. Dow, Carly K. Schissel…·Bradley L. Pentelute

This paper develops a high-throughput platform to discover arginine-depleted cell-penetrating peptides for improved antisense delivery in Duchenne muscular dystrophy, showing reduced toxicity and enhanced efficacy.

1 citation

mousedeep learning

𝕏 PostBluesky

NeuroscienceGliaPeer-reviewedDec 2, 2025

Astrocyte MCT1 expression does not contribute to the axonal degenerative phenotype observed with ubiquitous MCT1 depletion.

Thomas Philips, Emily G. Thompson, Olivia Spead…·Jeffrey D. Rothstein

This study shows that astrocyte MCT1 depletion does not cause axonal degeneration, unlike oligodendrocyte MCT1 depletion, suggesting astrocyte MCT1 is not essential for axonal energy homeostasis with aging.

DOI 10.1002/glia.70113

mouseexperimental assay

𝕏 PostBluesky

NeurosciencebioRxivPreprintAug 27, 2024

A robust evaluation of TDP-43, poly GP, cellular pathology, and behavior in a AAV-C9ORF72 (G4C2)66 mouse model

Emily G. Thompson, Olivia Spead, Sigvard Åkerman…·Jeffrey D. Rothstein

This study evaluates a mouse model of C9-ALS/FTD, finding it robust for molecular mechanisms but not for neurodegeneration and motor deficits.

DOI 10.1101/2024.08.27.607409

mouseexperimental assay

𝕏 PostBluesky