Author

Sean Miller

Johns Hopkins UniversityORCID 0000-0003-1432-9547Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Sean Miller1 paper · University of Notre Dame

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Antonino CattaneoiD· 1 shared Balaji G. Vijayakumar· 1 shared Brian P. HafleriD· 1 shared Emily G. ThompsoniD· 1 shared Erica R. Kent· 1 shared Francesca MalerbaiD· 1 shared Gerard WalkeriD· 1 shared Harry JungiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Antonino CattaneoORCID

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2 papers

Recent·Most cited

NeuroscienceGliaPeer-reviewedDec 2, 2025

Astrocyte MCT1 expression does not contribute to the axonal degenerative phenotype observed with ubiquitous MCT1 depletion.

Thomas Philips, Emily G. Thompson, Olivia Spead…·Jeffrey D. Rothstein

This study shows that astrocyte MCT1 depletion does not cause axonal degeneration, unlike oligodendrocyte MCT1 depletion, suggesting astrocyte MCT1 is not essential for axonal energy homeostasis with aging.

DOI 10.1002/glia.70113

mouseexperimental assay

𝕏 PostBluesky

NeurosciencebioRxivPreprintNov 18, 2025

A painless nerve growth factor variant uncouples nociceptive and neurotrophic TrkA signaling

Shailesh Kumar, Harry Jung, Gerard Walker…·Moitrayee Bhattacharyya

This paper identifies a NGF variant that selectively biases TrkA signaling towards neurotrophic functions without triggering pain, revealing mechanisms for therapeutic targeting of the NGF-TrkA pathway.

1 citation

humanexperimental assay

𝕏 PostBluesky