Author

Hervé Nicoloff

Uppsala UniversityORCID 0000-0002-4848-1371Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:E. coli· 3 Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Assay:whole-genome sequencing· 2 Research Focus:cell type annotation· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Dan I. AnderssoniD· 3 shared Enrique JoffréiD· 2 shared Andrei S. GuliaeviD· 1 shared Angelika NtokakiiD· 1 shared Bart Melgers· 1 shared Caroline Sentell· 1 shared Elin SvedholmiD· 1 shared Erik HolmqvistiD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersMicrobiology

Dan I. AnderssonORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Distinct plasmid- and host-encoded mechanisms drive small plasmid copy number-mediated heteroresistance in Escherichia coli

Angelika Ntokaki, Enrique Joffré, Bart Melgers, Margarita Komi, Erik Holmqvist, Dan I. Andersson, Hervé Nicoloff, Helen H. Wang

The paper shows small plasmids can cause antibiotic heteroresistance through copy number increases via plasmid mutations or chromosomal recD mutations in Escherichia coli.

bioRxiv · Aug 19, 2026 · PreprintMicrobiology

Novel phage-plasmid mediated mechanism of antibiotic heteroresistance in Escherichia coli

Elin Svedholm, Enrique Joffré, Caroline Sentell, Helen Wang, Dan I. Andersson, Hervé Nicoloff

This study identifies a novel mechanism of antibiotic heteroresistance in E. coli mediated by phage-plasmid copy number increase and horizontal transfer.

bioRxiv · Aug 19, 2026 · PreprintMicrobiology

Heteroresistance in Enterococcus faecalis is prevalent for key antibiotics and mainly caused by mutations

Sheida Heidarian, Sarah Lohsen, Andrei S. Guliaev, Hervé Nicoloff, Sarah W. Satola, Dan I. Andersson, Karin Hjort

This study identifies heteroresistance in Enterococcus faecalis against three antibiotics, showing that it is mainly caused by chromosomal mutations and mobile genetic elements.

bioRxiv · Jul 16, 2026 · PreprintMicrobiology