Author

Lifang Wang

BGI Group (China)ORCID 0000-0002-4936-4652Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Lifang Wang2 papers · BGI Group (China)

Biology Disciplines

Click to filter author papers

Neuroscience· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Assay:spatial transcriptomics· 2 Approach:statistical model· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:snRNA-seq· 1

Frequent Co-Authors

Lei HaniD· 2 shared Youzhe HeiD· 2 shared Aiqing LiiD· 1 shared Bufan Jin· 1 shared Cai SongiD· 1 shared De YangiD· 1 shared Dong ZhouiD· 1 shared Hao WangiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersNeuroscience

Lei HanORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Single-cell spatial transcriptomics and snRNA-seq decoding the organizational principles of functional modules in the mouse amygdala

Yu Bin, Xuan Chen, Yi-Heng Xu, Youzhe He, Lifang Wang, Zihan Wu, Jianhua Yao, Shuqi Liu, De Yang, Shuxia Cao, Hao Wang, Lei Han, Xiao-Ming Li

This study combines spatial transcriptomics and snRNA-seq to map cell type organization and functional modules in the mouse amygdala, revealing developmental origins and spatial patterns.

bioRxiv · Jun 5, 2026 · PreprintNeuroscience

Single-cell spatial transcriptome reveals pathological features of human hippocampus with sclerosis

Lifang Wang, Xingjie Li, Yuyang Liu, Bufan Jin, Yanrong Wei, Youzhe He, Yaru Ma, Zi-Han Wu, Xue Gong, Yanru An, Xueying Kong, Yanan Xing, Ruixi Ye, Quyuan Tao, Aiqing Li, , Zhen Yi Hong

This study uses single-cell spatial transcriptomics to identify molecular and cellular changes in hippocampal sclerosis associated with MTLE, revealing vulnerable subregions and novel pathological features.

bioRxiv · Jan 21, 2025 · PreprintNeuroscience