Prathibha Ranganathan

This study shows collagen in the tumor microenvironment promotes androgen independence in prostate cancer by enhancing cell migration, reducing chemotherapy sensitivity, and activating androgen receptor signaling through FAK.

This study identifies unique and differentially expressed proteins in cancer-associated fibroblasts from prostate cancer to uncover potential therapeutic targets.

This study compares the transcriptomes of normal and cancer-associated fibroblasts to identify differentially expressed genes that could inform therapeutic strategies.