Author

Katherine Luzuriaga

University of Massachusetts Chan Medical SchoolORCID 0000-0002-5561-1787Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 2 Bioinformatics· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Approach:machine learning (classical)· 1

Frequent Co-Authors

Anna GiliD· 2 shared Dario GhersiiD· 2 shared Larisa KamgaiD· 2 shared Inyoung Song· 1 shared Krishna Kumar GantaiD· 1 shared Lawrence J. Stern· 1 shared Liisa K. Selin· 1 shared Liisa Kaarina SeliniD· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersBioinformatics

Anna GilORCID

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3 papers

Recent·Most cited

ImmunologyCell ReportsPeer-reviewedMay 1, 2025

Acute infectious mononucleosis generates persistent, functional EBNA-1 antibodies with high cross-reactivity to alpha crystalline beta

Krishna Kumar Ganta, Margaret M. McManus, Ross Blanc…·Katherine Luzuriaga

The paper investigates EBNA-1 specific antibodies in IM patients, finding persistent functional antibodies with cross-reactivity to CRYAB and association with MS risk alleles.

7 citations

humanproteomics

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BioinformaticsBMC BioinformaticsPeer-reviewedSep 7, 2021

SwarmTCR: a computational approach to predict the specificity of T Cell Receptors

Ryan Ehrlich, Larisa Kamga, Anna Gil…·Dario Ghersi

SwarmTCR predicts T cell receptor specificity using a nearest-neighbor approach, outperforming TCRdist on specific epitopes with single-cell and bulk data.

23 citations

machine learning (classical)

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ImmunologybioRxivPreprintSep 29, 2018

High diversity, turnover, and structural constraints characterize TCR α and β repertoire selection

Larisa Kamga, Anna Gil, Inyoung Song…·Katherine Luzuriaga

The paper examines TCR α and β repertoire dynamics during EBV infection, finding that persistent clonotypes are highly expanded in acute infection and replaced by new clonotypes in convalescence.

DOI 10.1101/428623

humanproteomics

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