Author

Samuel Mackenzie

University of Rochester MedicineORCID 0000-0002-5571-7370Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

Alison Hobbins· 1 shared Angela StoutiD· 1 shared Harris A. GelbardiD· 1 shared Jennetta W. HammondiD· 1 shared Jenny M Gunnersen· 1 shared John Randolph· 1 shared Julia M. GranatoiD· 1 shared Nina Silver· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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1 shared paperNeuroscience

Alison Hobbins

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1 paper

Recent·Most cited

Loss of SEZ6L2 disrupts synaptic architecture and drives progressive behavioral deficits

Julia M. Granato, Nina Silver, Alison Hobbins, John Randolph, Angela Stout, Harris A. Gelbard, Jenny M Gunnersen, Samuel Mackenzie, Patricia M. White, Jennetta W. Hammond

This study characterizes Sez6l2 knockout mice, showing behavioral and synaptic deficits, and compares them to Sez6 triple knockout mice, highlighting SEZ6L2's non-redundant role in brain function.

bioRxiv · Sep 24, 2026 · PreprintNeuroscience