Author

Adi Inbal

Hebrew University of JerusalemORCID 0000-0002-5666-109XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:zebrafish· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Ann SaadaiD· 2 shared Shira Yanovsky‐DaganiD· 2 shared Shlomit EzeriD· 2 shared Tamar HareliD· 2 shared Amit Granit· 1 shared Hadar Benyamini· 1 shared Inbar PlaschkesiD· 1 shared Israel Antman· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersGenetics

Ann SaadaORCID

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2 papers

Recent·Most cited

Allele-specific antisense oligonucleotide treatment rescues atad3-associated phenotype in zebrafish

Shlomit Ezer, Shira Yanovsky‐Dagan, Amit Granit, Matthew McDougal, Taeyoung Hwang, Israel Antman, Rotem Karni, Wan Hee Yoon, Ann Saada, Adi Inbal, Tamar Harel

This study demonstrates that allele-specific antisense oligonucleotides can rescue ATAD3A-associated developmental defects in zebrafish by reducing mutant transcripts.

bioRxiv · May 23, 2026 · PreprintGenetics

Transcriptome analysis of atad3-null zebrafish embryos elucidates possible disease mechanisms

Shlomit Ezer, Nathan Ronin, Shira Yanovsky‐Dagan, Shahar Rotem‐Bamberger, Orli Halstuk, Yair Wexler, Zohar Ben-Moshe, Inbar Plaschkes, Hadar Benyamini, Ann Saada, Adi Inbal, Tamar Harel

This study uses zebrafish atad3 knockout models to identify mitochondrial dysfunction as a key mechanism in ATAD3A-related human diseases.

Orphanet Journal of Rare Diseases · Apr 15, 2025Peer-reviewedGenetics