Author

Hadar Benyamini

Hebrew University of Jerusalem

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 1 Genetics· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Organism:human· 1

Frequent Co-Authors

Adi InbaliD· 1 shared Alexander Henderson· 1 shared Andrew Nutter-Upham· 1 shared Ann SaadaiD· 1 shared Antony A. CooperiD· 1 shared Autumn Rose MorrowiD· 1 shared Ayelet Kohl· 1 shared Camila A. ZanellaiD· 1 shared

Activity by Year

2024-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperGenetics

Adi InbalORCID

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Author profile →

3 papers

Recent·Most cited

Transcriptome analysis of atad3-null zebrafish embryos elucidates possible disease mechanisms

Shlomit Ezer, Nathan Ronin, Shira Yanovsky‐Dagan, Shahar Rotem‐Bamberger, Orli Halstuk, Yair Wexler, Zohar Ben-Moshe, Inbar Plaschkes, Hadar Benyamini, Ann Saada, Adi Inbal, Tamar Harel

This study uses zebrafish atad3 knockout models to identify mitochondrial dysfunction as a key mechanism in ATAD3A-related human diseases.

Orphanet Journal of Rare Diseases · Apr 15, 2025Peer-reviewedGenetics

Deep sequencing of proteotoxicity modifier genes uncovers a Presenilin-2/beta-amyloid-actin genetic risk module shared among alpha-synucleinopathies

Sumaiya Nazeen, Xinyuan Wang, Dina Zielinski, Isabel Lam, Erinc Hallacli, Ping Xu, Elizabeth Ethier, Ronya Strom, Camila A. Zanella, Vanitha Nithianandam, Dylan Ritter, Alexander Henderson, Nathalie G. Saurat, Jalwa Afroz, Andrew Nutter-Upham, , Vikram Khurana

The paper identifies a shared genetic risk module involving PSEN2 and actin regulation across alpha-synucleinopathies, linking them to Alzheimer's disease mechanisms.

bioRxiv · Mar 3, 2024 · PreprintGenomics

Hindbrain boundaries as niches of neural progenitor/stem cells regulated by the extracellular matrix proteoglycan chondroitin sulphate.

Carmel Hutchings, Yarden Nuriel, Daniel Lazar, Ayelet Kohl, Elizabeth M. Muir, Olga Genin, Yuval Cinnamon, Hadar Benyamini, Yuval Nevo, Dalit Sela‐Donenfeld

The paper identifies hindbrain boundaries as niches containing neural progenitor/stem cells regulated by chondroitin sulphate proteoglycan through transcriptomic analysis.

Development · Jan 22, 2024Peer-reviewedDevelopmental Biology