Author

Hiroki Kurahashi

Fujita Health UniversityORCID 0000-0002-5690-5218Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Ewelina Bolcun‐FilasiD· 1 shared Hiroshi KogoiD· 1 shared John C. SchimentiiD· 1 shared Kazumi KimuraiD· 1 shared Kazuo YamamotoiD· 1 shared Masakazu Souda· 1 shared Masashi AsaiiD· 1 shared Ryotaro MoriiD· 1 shared

Activity by Year

2017-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperGenetics

Ewelina Bolcun‐FilasORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Identification of the Down syndrome critical region 3 gene as a mammalian cell size regulator

Kazumi Kimura, Masakazu Souda, Ryotaro Mori, Yoshimi Kato, Hiroki Kurahashi, Masashi Asai, Kazuo Yamamoto

This study identifies DSCR3 as a gene that regulates cell size, showing its overexpression increases cell size and its knockdown reduces it in fibroblasts.

Genes to Cells · Aug 14, 2026Peer-reviewedCell Biology

A Single Checkpoint Pathway Eliminates Mouse Oocytes With DNA Damage Or Chromosome Synapsis Failure

Vera D. Rinaldi, Ewelina Bolcun‐Filas, Hiroshi Kogo, Hiroki Kurahashi, John C. Schimenti

The paper shows that the CHK2-dependent checkpoint eliminates mouse oocytes with DNA damage or synapsis failure by detecting and removing those with unrepaired DSBs or unsynapsed chromosomes.

Molecular Cell · Aug 24, 2017Peer-reviewedGenetics