Author

Hiroshi Kogo

Gunma UniversityORCID 0000-0003-0584-7158Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Assay:imaging· 1

Frequent Co-Authors

Akiko Iizuka‐KogoiD· 1 shared Ewelina Bolcun‐FilasiD· 1 shared Hanako Yamamoto· 1 shared Hiroki KurahashiiD· 1 shared John C. SchimentiiD· 1 shared Maiko IkezawaiD· 1 shared Toshiyuki MatsuzakiiD· 1 shared Vera D. RinaldiiD· 1 shared

Activity by Year

2017-2025 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 16 mutual co-author links

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1 shared paperDevelopmental Biology

Akiko Iizuka‐KogoORCID

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2 papers

Recent·Most cited

Developmental BiologyHistochemistry and Cell BiologyPeer-reviewedFeb 17, 2025

A simple immunohistochemical method for perinatal mammalian ovaries revealed different kinetics of oocyte apoptosis caused by DNA damage and asynapsis.

Hiroshi Kogo, Akiko Iizuka‐Kogo, Hanako Yamamoto…·Toshiyuki Matsuzaki

The paper introduces a simple squash method for immunohistochemical analysis of perinatal mouse ovaries, revealing different kinetics of oocyte apoptosis caused by DNA damage and asynapsis.

1 citation

mouseimaging

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GeneticsMolecular CellPeer-reviewedAug 24, 2017

A Single Checkpoint Pathway Eliminates Mouse Oocytes With DNA Damage Or Chromosome Synapsis Failure

Vera D. Rinaldi, Ewelina Bolcun‐Filas, Hiroshi Kogo…·John C. Schimenti

The paper shows that the CHK2-dependent checkpoint eliminates mouse oocytes with DNA damage or synapsis failure by detecting and removing those with unrepaired DSBs or unsynapsed chromosomes.

131 citationstop 4%

mouseDNA methylation

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